Impaired mitochondrial function, oxidative stress and altered antioxidant enzyme activities following traumatic spinal cord injury
- 1 August 1997
- journal article
- Published by Elsevier in Brain Research
- Vol. 765 (2) , 283-290
- https://doi.org/10.1016/s0006-8993(97)00573-8
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- Long term treatment with (−)deprenyl reduces the optimal dose as well as the effective dose range for increasing antioxidant enzyme activities in old mouse brainLife Sciences, 1996
- Structural and functional analogs of CuZn superoxide dismutase inhibit rat brain nitric oxide synthase by interference with the reductase (diaphorase) domainNeuroscience Letters, 1996
- Calcium Movements in Traumatic Brain Injury: The Role of Glutamate Receptor-Operated Ion ChannelsJournal of Cerebral Blood Flow & Metabolism, 1996
- SOD‐1 Transgenic Mice as a Model for Studies of Neuroprotection in Stroke and Brain TraumaaAnnals of the New York Academy of Sciences, 1994
- Glucocorticoid Therapy of Spinal Cord InjuryaAnnals of the New York Academy of Sciences, 1994
- Pathophysiology of spinal cord traumaAnnals of Emergency Medicine, 1993
- Lipid antioxidants in acute central nervous system injuryAnnals of Emergency Medicine, 1993
- Regional Changes in Interstitial K+and Ca2+Levels following Cortical Compression Contusion Trauma in RatsJournal of Cerebral Blood Flow & Metabolism, 1993
- 21-Aminosteroids attenuate excitotoxic neuronal injury in cortical cell culturesNeuron, 1990
- A Randomized, Controlled Trial of Methylprednisolone or Naloxone in the Treatment of Acute Spinal-Cord InjuryNew England Journal of Medicine, 1990