Contribution of the renin–angiotensin system to subsensitivity of soluble guanylyl cyclase in TGR(mREN2)27 rats
- 28 August 2000
- journal article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 403 (1-2) , 27-35
- https://doi.org/10.1016/s0014-2999(00)00577-x
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Loss of Expression of the β Subunit of Soluble Guanylyl Cyclase Prevents Nitric Oxide–Mediated Inhibition of DNA Synthesis in Smooth Muscle Cells of Old RatsCirculation Research, 2000
- Expression of nitric oxide-sensitive guanylyl cyclase subunits in human corpus cavernosumBiochemical Pharmacology, 2000
- Increased Nitrovasodilator Sensitivity in Endothelial Nitric Oxide Synthase Knockout MiceHypertension, 2000
- Endothelial Dysfunction in Chronic Myocardial Infarction Despite Increased Vascular Endothelial Nitric Oxide Synthase and Soluble Guanylate Cyclase ExpressionCirculation, 1999
- Pulsatile Stretch and Shear Stress: Physical Stimuli Determining the Production of Endothelium-Derived Relaxing FactorsJournal of Vascular Research, 1998
- The beta2 subunit inhibits stimulation of the alpha1/beta1 form of soluble guanylyl cyclase by nitric oxide. Potential relevance to regulation of blood pressure.Journal of Clinical Investigation, 1997
- Nitric oxide decreases stability of mRNAs encoding soluble guanylate cyclase subunits in rat pulmonary artery smooth muscle cells.Journal of Clinical Investigation, 1997
- Nitric Oxide and Its Putative Role in HypertensionHypertension, 1995
- Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells.Circulation Research, 1994
- Transgenic rats carrying the mouse renin gene—Morphological characterization of a low-renin hypertension modelKidney International, 1992