The molecular pathophysiology of pain: abnormal expression of sodium channel genes and its contributions to hyperexcitability of primary sensory neurons
- 1 August 1999
- journal article
- review article
- Published by Wolters Kluwer Health in Pain
- Vol. 82 (Supplement) , S133-S140
- https://doi.org/10.1016/s0304-3959(99)00147-5
Abstract
Although hyperexcitability and/or increased baseline sensitivity of primary sensory neurons following nerve injury can lead to abnormal burst activity associated with pain, the molecular mechanisms that contribute to it are not fully understood. Early studies demonstrated that, following axoKeywords
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