Cardiac work up in primary renal hypokalaemia-hypomagnesaemia (Gitelman syndrome)
Open Access
- 19 March 2004
- journal article
- Published by Oxford University Press (OUP) in Nephrology Dialysis Transplantation
- Vol. 19 (6) , 1398-1402
- https://doi.org/10.1093/ndt/gfh204
Abstract
Background. Potassium and magnesium depletion prolongs the duration of the action potential of the cardiomyocyte, which predisposes to ventricular arrhythmias. In addition, potassium or magnesium depletion might impair cardiac performance and facilitate coronary artery thrombosis.Keywords
This publication has 22 references indexed in Scilit:
- The biochemical diagnosis of Gitelman disease and the definition of "hypocalciuria"Pediatric Nephrology, 2003
- Electrocardiogram with prolonged QT interval in Gitelman diseaseKidney International, 2002
- Two novel mutations and a new STK11/LKB1 gene isoform in Peutz-Jeghers patientsHuman Mutation, 2002
- Clinical and therapeutic aspects of congenital and acquired long QT syndromeThe American Journal of Medicine, 2002
- Cardiovascular Actions of MagnesiumCritical Care Clinics, 2001
- Bartter's Syndrome Comes of AgePublished by American Academy of Pediatrics (AAP) ,1999
- Gitelman syndrome comes of ageNephrology Dialysis Transplantation, 1998
- Molecular Biological Methods for Characterizing Drug-Metabolizing Enzymes in Hepatic and Extrahepatic TissuesSkin Pharmacology and Physiology, 1998
- Genetic heterogeneity in tubular hypomagnesemia-hypokalemia with hypocalcuria (Gitelman's syndrome)Kidney International, 1995
- Use of calcium excretion values to distinguish two forms of primary renal tubular hypokalemic alkalosis: Bartter and Gitelman syndromesThe Journal of Pediatrics, 1992