LYMPHOCYTE SUBPOPULATIONS AT THE SITE OF PIECEMEAL NECROSIS IN END STAGE CHRONIC LIVER-DISEASES AND REJECTING LIVER ALLOGRAFTS IN CYCLOSPORINE-TREATED PATIENTS
- 1 January 1984
- journal article
- research article
- Vol. 50 (3) , 341-347
Abstract
Mononuclear cells in the areas of piecemeal necrosis in hepatic tissues from patients with different liver diseases were subtyped using monoclonal antibodies and the avidin-biotin-peroxidase complex method. T lymphocytes were the predominant infiltrating cell type in this lesion (> 75% of mononuclear cells) regardless of the etiology of the liver disease. M1-positive cells represented .apprx. 20% of the infiltrate. B cells were absent and rare Leu-7-positive cells (killer, natural killer) showed random tissue distribution in both the periportal areas and the parenchyma. The T8+ (suppressor-cytotoxic) lymphocytes accumulated selectively in the areas of piecemeal necrosis whereas the tissue T4/T8 ratios were consistently < 1. T8+ lymphocytes were also more numerous than T4+ cells around the hepatitis B surface antigen-positive hepatocytes in piecemeal necrotic areas of livers from patients with hepatitis B surface antigen-positive chronic active hepatitis. In liver tissues of patients with primary biliary cirrhosis, alcoholic cirrhosis, chronic active hepatitis and rejecting liver allografts, selective accumulations of T8+ lymphocytes at the sites of hepatocyte necrosis were a characteristic and uniform finding. This subpopulation of lymphocytes may be an important part of the immunologically mediated destruction of hepatocytes that occurs in chronic active liver diseases and is characterized by piecemeal necrosis.This publication has 12 references indexed in Scilit:
- Tissue distribution of human NK cells studied with anti-Leu-7 monoclonal antibody.The Journal of Immunology, 1983
- A QUANTITATIVE ULTRASTRUCTURAL ANALYSIS OF THE LEUKOCYTES IN CONTACT WITH HEPATOCYTES IN CHRONIC ACTIVE HEPATITIS, WITH A CYTOCHEMICAL DETECTION OF MONONUCLEAR PHAGOCYTES1982
- CELLULAR AND HUMORAL IMMUNE-REACTIONS IN CHRONIC ACTIVE LIVER-DISEASE .1. LYMPHOCYTE SUBSETS IN LIVER BIOPSIES OF PATIENTS WITH UNTREATED IDIOPATHIC AUTOIMMUNE HEPATITIS, CHRONIC ACTIVE HEPATITIS-B AND PRIMARY BILIARY-CIRRHOSIS1982
- Suppression of endogenous avidin-binding activity in tissues and its relevance to biotin-avidin detection systems.Journal of Histochemistry & Cytochemistry, 1981
- Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.Journal of Histochemistry & Cytochemistry, 1981
- DETECTION OF TISSUE T-CELL IN PATIENTS WITH CHRONIC ACTIVE HEPATITIS USING FRAGMENTED SHEEP RED-BLOOD-CELL (SRBC) MEMBRANE1980
- HEPATOCYTE DAMAGE INDUCED BY LYMPHOCYTES FROM PATIENTS WITH CHRONIC LIVER-DISEASES, AS DETECTED BY LDH RELEASE1978
- MORPHOLOGIC ASSOCIATION OF LYMPHOCYTES WITH HEPATOCYTES IN CHRONIC LIVER-DISEASE1977
- Chronic hepatitis: a problem for the pathologistHistopathology, 1977
- ANTIBODY-DEPENDENT CELL-MEDIATED (K CELL) CYTOTOXICITY AGAINST ISOLATED HEPATOCYTES IN CHRONIC ACTIVE HEPATITISThe Lancet, 1976