Antisense oligodeoxynucleotides directed against aspartyl (asparaginyl) β-hydroxylase suppress migration of cholangiocarcinoma cells
- 31 May 2003
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 38 (5) , 615-622
- https://doi.org/10.1016/s0168-8278(03)00052-7
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- The Role of P27kip1 Protein Expression on the Biological Behavior of Intrahepatic CholangiocarcinomaHepatology, 2001
- K-ras mutations in the bile of patients with primary sclerosing cholangitisGut, 2001
- Expression and Antitumor Effects of TRAIL in Human CholangiocarcinomaHepatology, 2000
- Biliary Tract CancersNew England Journal of Medicine, 1999
- Yeast polypeptide fusion surface display levels predict thermal stability and soluble secretion efficiency 1 1Edited by J. A. WellsJournal of Molecular Biology, 1999
- Clinical use of streptolysin-O to facilitate antisense oligodeoxyribonucleotide delivery for purging autografts in chronic myeloid leukaemiaBone Marrow Transplantation, 1999
- The five cysteine residues located in the active site region of bovine aspartyl (asparaginyl) β-hydroxylase are not essential for catalysisBiochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 1998
- Hepatocytic phenotypes induced in sarcomatous cholangiocarcinoma cells treated with 5-azacytidineHepatology, 1997
- Enhanced Antisense Effects Resulting from an Improved Streptolysin-O Protocol for Oligodeoxynucleotide Delivery into Human Leukaemia CellsNucleosides and Nucleotides, 1997
- Invertebrate aspartyl/asparaginyl β-hydroxylase: Potential modification of endogenous epidermal growth factor-like modulesBiochemical and Biophysical Research Communications, 1992