Particulate Matter Initiates Inflammatory Cytokine Release by Activation of Capsaicin and Acid Receptors in a Human Bronchial Epithelial Cell Line
- 1 January 1999
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 154 (1) , 106-115
- https://doi.org/10.1006/taap.1998.8567
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- Evidence That Exposure of Particulate Air Pollutants to Human and Rat Alveolar Macrophages Leads to Differential Oxidative ResponseBiochemical and Biophysical Research Communications, 1997
- SOLUBLE TRANSITION METALS MEDIATE RESIDUAL OIL FLY ASH INDUCED ACUTE LUNG INJURYJournal of Toxicology and Environmental Health, 1997
- Role of tachykinins and neutral endopeptidase in toluene diisocyanate-induced bronchial hyperresponsiveness in guinea pigsToxicology, 1997
- Induction of vanadium accumulation and nuclear sequestration causing cell suicide in human Chang liver cellsCellular and Molecular Life Sciences, 1996
- Vanadium-Induced Chemokine mRNA Expression and Pulmonary InflammationToxicology and Applied Pharmacology, 1996
- A Novel System for the in Vitro Exposure of Pulmonary Cells to Acid Sulfate AerosolsFundamental and Applied Toxicology, 1993
- Airway hyperresponsiveness in guinea pigs exposed to acid‐coated ultrafine particlesJournal of Toxicology and Environmental Health, 1992
- Suppression of alveolar macrophage membrane-receptor-mediated phagocytosis by model particle-adsorbate complexes: physicochemical moderators of uptake.Environmental Health Perspectives, 1990
- The proton-activated inward current of rat sensory neurons includes a calcium componentNeuroscience Letters, 1990
- Compound forms of fossil fuel fly ash emissionsEnvironmental Science & Technology, 1980