Excess Copper and Ceruloplasmin Biosynthesis in Long-term Cultured Hepatocytes from Long-Evans Cinnamon (LEC) Rats, a Model of Wilson Disease
Open Access
- 1 March 1995
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 270 (13) , 7656-7660
- https://doi.org/10.1074/jbc.270.13.7656
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- The LEC rat has a deletion in the copper transporting ATPase gene homologous to the Wilson disease geneNature Genetics, 1994
- Isolation and Characterization of a Human Liver cDNA as a Candidate Gene for Wilson DiseaseBiochemical and Biophysical Research Communications, 1993
- Genetic linkage between copper accumulation and hepatitis/hepatoma development in LEC ratsMolecular Carcinogenesis, 1992
- Spontaneous hepatic copper accumulation in Long-Evans Cinnamon rats with hereditary hepatitis. A model of Wilson's disease.Journal of Clinical Investigation, 1991
- Angiogenic FactorsScience, 1987
- Binding and uptake of copper from ceruloplasminBiochemical and Biophysical Research Communications, 1986
- Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications.Proceedings of the National Academy of Sciences, 1979
- Reinvestigation of some physicochemical and chemical properties of human ceruloplasmin (ferroxidase)Biochemistry, 1976
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970
- Ceruloplasmin in Wilson's Disease*Journal of Clinical Investigation, 1967