Abstract
Histamine, 4-methylhistamine, 3-(β-aminoethyl)-1,2,4 triazole and betazole, in that order, stimulated adenylate cyclase prepared from rat gastric tissue in a dose-dependent manner. Burimamide, an H2-receptor blocking agent, in concentrations of 1–5 × 10−6 m antagonized this effect. The data lend some support to the hypothesis that elevated levels of cyclic adenosine 3′,5′-monophosphate may be involved in histamine-stimulated gastric secretions and that H2-receptors are associated with adenylate cyclase.

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