Depolarization inactivation of dopamine neurons: Terminal release characteristics

This publication has 20 references indexed in Scilit:

• The effects of dopamine-depleting brain lesions on the electrophysiological activity of rat substantia nigra dopamine neurons
  Brain Research, 1990
• Differential Effect of Stress on In Vivo Dopamine Release in Striatum, Nucleus Accumbens, and Medial Frontal Cortex
  Journal of Neurochemistry, 1989
• Haloperidol given chronically decreases basal dopamine in the prefrontal cortex more than the striatum or nucleus accumbens as simultaneously measured by microdialysis
  Brain Research Bulletin, 1989
• Nonlinear relationship between impulse flow and dopamine released by rat midbrain dopaminergic neurons as studied by in vivo electrochemistry
  Neuroscience, 1988
• Tolerance to haloperidol-induced increases in dopamine metabolites: fact or artifact?
  European Journal of Pharmacology, 1987
• Regulation of dopamine release by impulse flow and by autoreceptors as studied by in vivo voltammetry in the rat striatum
  Neuroscience, 1985
• Typical and atypical neuroleptics: differential effects of chronic administration on the activity of A9 and A10 midbrain dopaminergic neurons
  Journal of Neuroscience, 1983
• Unique response to antipsychotic drugs is due to absence of terminal autoreceptors in mesocortical dopamine neurones
  Nature, 1982
• Acute and chronic haloperidol treatment: Comparison of effects on nigral dopaminergic cell activity
  Life Sciences, 1978
• Tolerance to tyrosine hydroxylase activation in N. Accumbens and C. Striatum after repeated injections of “classical” and “atypical” antischizophrenic drugs
  Life Sciences, 1978