Different genes specify hyporesponsiveness to seizures induced by caffeine and the benzodiazepine inverse agonist, DMCM
- 1 July 1987
- journal article
- research article
- Published by Elsevier in Pharmacology Biochemistry and Behavior
- Vol. 27 (3) , 451-456
- https://doi.org/10.1016/0091-3057(87)90348-0
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Coincidence of seizure susceptibility to caffeine and to the benzodiazepine inverse agonist, DMCM, in SWR and CBA inbred micePharmacology Biochemistry and Behavior, 1987
- Inherent hyporesponsiveness to methylxanthine-induced behavioral changes associated with supersensitivity to 5'-N-ethylcarboxamidoadenosine (NECA)Pharmacology Biochemistry and Behavior, 1986
- Inherent differences in sensitivity to methylxanthines among inbred micePharmacology Biochemistry and Behavior, 1986
- Sensitivity of inbred mice to methylxanthines is not determined by plasma xanthine concentrationNeuroscience Letters, 1985
- Benzodiazepine receptor ligands with positive and negative efficacyNeuropharmacology, 1983
- Pharmacokinetic and pharmacodynamic factors contributing to the convulsant action of β-carboline-3-carboxylic acid estersLife Sciences, 1983
- DMCM: A potent convulsive benzodiazepine receptor ligandEuropean Journal of Pharmacology, 1983
- An allosteric model for benzodiazepine receptor functionBiochemical Pharmacology, 1983
- Binding of β‐Carbolines and Caffeine on Benzodiazepine Receptors: Correlations to Convulsions and TremorActa Pharmacologica et Toxicologica, 1982
- Purinergic inhibition of diazepam binding to rat brain ()Life Sciences, 1979