“Benign erythrocytosis” and other familial and congenital polycythemias
Open Access
- 1 October 1996
- journal article
- Published by Wiley in European Journal of Haematology
- Vol. 57 (4) , 263-268
- https://doi.org/10.1111/j.1600-0609.1996.tb01376.x
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Erythropoietin Induces the Tyrosine Phosphorylation, Nuclear Translocation, and DNA Binding of STAT1 and STAT5 in Erythroid CellsJournal of Biological Chemistry, 1995
- Familial PolycythemiaScience, 1995
- Specific recruitment of SH-PTP1 to the erythropoietin receptor causes inactivation of JAK2 and termination of proliferative signalsCell, 1995
- Erythropoietin receptor mRNA expression in human endothelial cells.Proceedings of the National Academy of Sciences, 1994
- Two microsatellite repeat polymorphisms in the EPO geneHuman Molecular Genetics, 1994
- Primary familial and congenital polycythaemiaThe Lancet, 1993
- JAK2 associates with the erythropoietin receptor and is tyrosine phosphorylated and activated following stimulation with erythropoietinCell, 1993
- Truncated erythropoietin receptor causes dominantly inherited benign human erythrocytosis.Proceedings of the National Academy of Sciences, 1993
- Expression cloning of the murine erythropoietin receptorCell, 1989
- Congenital Erythrocytosis: a New Form Associated with an Erythropoietin‐dependent MechanismBritish Journal of Haematology, 1980