Geranylgeranyl as Well as Farnesyl Moiety Is Transferred to Ras p21 Overproduced in Adrenocortical Cells Transformed by c-Ha-rasEJOncogene

This publication has 19 references indexed in Scilit:

• PROTEIN PRENYLATION: Molecular Mechanisms and Functional Consequences
  Annual Review of Biochemistry, 1996
• Resistance of K-RasBV12 proteins to farnesyltransferase inhibitors in Rat1 cells.
  Proceedings of the National Academy of Sciences, 1996
• Disruption of Oncogenic K-Ras4B Processing and Signaling by a Potent Geranylgeranyltransferase I Inhibitor
  Published by Elsevier ,1995
• Specific Labeling of Isoprenylated Proteins: Application to Study Inhibitors of the Post-translational Farnesylation and Geranylgeranylation
  Biochemical and Biophysical Research Communications, 1995
• Role of Protein Modification Reactions in Programming Interactions Between Ras-Related GTPases and Cell Membranes
  Annual Review of Cell Biology, 1994
• Benzodiazepine Peptidomimetics: Potent Inhibitors of Ras Farnesylation In Animal Cells
  Science, 1993
• PROTEIN ISOPRENYLATION AND METHYLATION AT CARBOXYL-TERMINAL CYSTEINE RESIDUES
  Annual Review of Biochemistry, 1992
• Aldosterone biosynthesis induced by ACTH and angiotensin II in newborn rat adrenocortical cells transfected by c-EJ-Ha-ras oncogene
  Biochemical and Biophysical Research Communications, 1991
• A cellular oncogene (c-Ki-ras) is amplified, overexpressed, and located within karyotypic abnormalities in mouse adrenocortical tumour cells
  Nature, 1983
• Mechanism of activation of a human oncogene
  Nature, 1982