Differences in the early inflammatory responses to toxin-induced demyelination are associated with the age-related decline in CNS remyelination
- 26 July 2005
- journal article
- research article
- Published by Elsevier in Neurobiology of Aging
- Vol. 27 (9) , 1298-1307
- https://doi.org/10.1016/j.neurobiolaging.2005.06.008
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Increased expression of Nkx2.2 and Olig2 identifies reactive oligodendrocyte progenitor cells responding to demyelination in the adult CNSMolecular and Cellular Neuroscience, 2004
- Functional Genomic Analysis of Remyelination Reveals Importance of Inflammation in Oligodendrocyte RegenerationJournal of Neuroscience, 2003
- Decline in Rate of Colonization of Oligodendrocyte Progenitor Cell (OPC)-Depleted Tissue by Adult OPCs with AgeJournal of Neuropathology and Experimental Neurology, 2003
- Efficient central nervous system remyelination requires T cellsAnnals of Neurology, 2003
- Growth factor treatment promotes mobilization of young but not aged adult subventricular zone precursors in response to demyelinationJournal of Neuroscience Research, 2002
- Premyelinating Oligodendrocytes in Chronic Lesions of Multiple SclerosisNew England Journal of Medicine, 2002
- The Protective Role of Nitric Oxide in a Neurotoxicant- Induced Demyelinating ModelThe Journal of Immunology, 2002
- TNFα promotes proliferation of oligodendrocyte progenitors and remyelinationNature Neuroscience, 2001
- Stimulation of oligodendroglial proliferation and maturation by interleukin-2Nature, 1986