Novel N-thiolated β-lactam antibiotics selectively induce apoptosis in human tumor and transformed, but not normal or nontransformed, cells
- 1 January 2004
- journal article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 67 (2) , 365-374
- https://doi.org/10.1016/j.bcp.2003.09.017
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Apoptosis SignalingAnnual Review of Biochemistry, 2000
- Biochemical Pathways of Caspase Activation During ApoptosisAnnual Review of Cell and Developmental Biology, 1999
- Studies on Nonconventionally Fused Bicyclic β-LactamsThe Journal of Organic Chemistry, 1998
- Degradation of Retinoblastoma Protein in Tumor Necrosis Factor- and CD95-induced Cell DeathPublished by Elsevier ,1997
- Yama/CPP32β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymeraseCell, 1995
- Apoptosis in the Pathogenesis and Treatment of DiseaseScience, 1995
- Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICENature, 1994
- Mechanisms and Functions of Cell DeathAnnual Review of Cell Biology, 1991
- Recent Progress in the Chemical Synthesis of AntibioticsPublished by Springer Nature ,1990
- Discovery and development of new β-lactam antibioticsPublished by Walter de Gruyter GmbH ,1987