Iron-Dependent Self-Assembly of Recombinant Yeast Frataxin: Implications for Friedreich Ataxia
- 1 September 2000
- journal article
- Published by Elsevier in American Journal of Human Genetics
- Vol. 67 (3) , 549-562
- https://doi.org/10.1086/303056
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- CCC1 Suppresses Mitochondrial Damage in the Yeast Model of Friedreich's Ataxia by Limiting Mitochondrial Iron AccumulationJournal of Biological Chemistry, 2000
- The ferritins: molecular properties, iron storage function and cellular regulationPublished by Elsevier ,1999
- Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxiaNature Genetics, 1997
- Studies of human, mouse and yeast homologues indicate a mitochondrial function for frataxinNature Genetics, 1997
- Regulation of Mitochondrial Iron Accumulation by Yfh1p, a Putative Homolog of FrataxinScience, 1997
- A kinetic study of iron release from Azotobacter vinelandii bacterial ferritinJournal of Inorganic Biochemistry, 1996
- Direct Observation of Enzyme Activity with the Atomic Force MicroscopeScience, 1994
- Biomolecular Imaging with the Atomic Force MicroscopeAnnual Review of Biophysics, 1994
- At least six nucleotides preceding the AUG initiator codon enhance translation in mammalian cellsJournal of Molecular Biology, 1987
- Mitochondrial iron not bound in heme and iron-sulfur centers and its availability for heme synthesis in vitroBiochimica et Biophysica Acta (BBA) - General Subjects, 1985