Inhibition of lipid peroxidation by ubiquinol in submitochondrial particles in the absence of vitamin E

Abstract
The relationship between the antioxidant effects of reduced coenzyme Q10 (ubiquinol, UQH2) and vitamin E (α‐tocopherol) was investigated in beef heart submitochondrial particles in which lipid peroxidation was initiated by incubation with ascorbate + ADP‐Fe3+. These effects were examined after extraction of coenzyme Q10 (UQ‐10) and vitamin E from the particles and reincorporation of the same components alone or in combination. The results show that UQHZ efficiently inhibits lipid peroxidation even when vitamin E is absent. It is concluded that UQH2 can inhibit lipid peroxidation directly, without the mediation of vitamin E.

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