Changes in Caspase Expression in Alzheimer's Disease: Comparison with Development and Aging
- 1 March 1999
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 256 (2) , 381-384
- https://doi.org/10.1006/bbrc.1999.0344
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- Differential Expression of Rat Brain Bcl-2 Family Proteins in Development and AgingBiochemical and Biophysical Research Communications, 1998
- Is Apoptosis Key in Alzheimer's Disease?Published by American Association for the Advancement of Science (AAAS) ,1998
- Alteration of proteins regulating apoptosis, Bcl-2, Bcl-x, Bax, Bak, Bad, ICH-1 and CPP32, in Alzheimer's diseaseBrain Research, 1998
- Life and Death of Neurons in the Aging BrainScience, 1997
- Apoptosis and Oxidative Stress in the Aging BrainAnnals of the New York Academy of Sciences, 1996
- The Bcl-2 family of proteins, and the regulation of neuronal survivalTrends in Neurosciences, 1995
- Bad, a heterodimeric partner for Bcl-xL and Bcl-2, displaces bax and promotes cell deathCell, 1995
- Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell deathCell, 1994
- The Consortium to Establish a Registry for Alzheimer's Disease (CERAD)Neurology, 1991
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976