Atypical mechanism of NF-κB activation during reoxygenation stress in microvascular endothelium: a role for tyrosine kinases
- 8 August 2002
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 33 (7) , 962-973
- https://doi.org/10.1016/s0891-5849(02)00990-5
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- The relationship between visceral ischemia, proinflammatory cytokines, and organ injury in patients undergoing thoracoabdominal aortic aneurysm repairCritical Care Medicine, 2000
- Pathophysiology of ischaemia-reperfusion injuryThe Journal of Pathology, 2000
- Treating myocardial ischemia-reperfusion injury by targeting endothelial cell transcriptionThe Annals of Thoracic Surgery, 1999
- A cytokine-responsive IκB kinase that activates the transcription factor NF-κBNature, 1997
- Identification and Characterization of an IκB KinaseCell, 1997
- Endothelial cell injury in cardiovascular surgery: The pathophysiology of vasomotor dysfunctionThe Annals of Thoracic Surgery, 1996
- Control of IκB-α Proteolysis by Site-Specific, Signal-Induced PhosphorylationScience, 1995
- Cytokine Patterns in Patients After Major Vascular Surgery, Hemorrhagic Shock, and Severe Blunt Trauma Relation with Subsequent Adult Respiratory Distress Syndrome and Multiple Organ FailureAnnals of Surgery, 1993
- NF-κB Controls Expression of Inhibitor IκBα: Evidence for an Inducible Autoregulatory PathwayScience, 1993
- Ischemia/reperfusion-induced changes in membrane fluidity characteristics of brain capillary endothelial cells and its prevention by liposomal-incorporated superoxide dismutaseBiochimica et Biophysica Acta (BBA) - Biomembranes, 1991