A pertussis/choleratoxin-sensitive N protein may mediate chemoattractant receptor signal transduction
- 1 July 1986
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 138 (2) , 887-894
- https://doi.org/10.1016/s0006-291x(86)80579-4
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Pertussis toxin inhibits chemotactic peptide-stimulated generation of inositol phosphates and lysosomal enzyme secretion in human leukemic (HL-60) cells.Proceedings of the National Academy of Sciences, 1985
- Pertussis toxin inhibits fMet-Leu-Phe- but not phorbol ester-stimulated changes in rabbit neutrophils: role of G proteins in excitation response coupling.Proceedings of the National Academy of Sciences, 1985
- Potential role for a guanine nucleotide regulatory protein in chemoattractant receptor mediated polyphosphoinositide metabolism, Ca++ mobilization and cellular responses by leukocytesBiochemical and Biophysical Research Communications, 1985
- Chemoattractant Receptors on Phagocytic CellsAnnual Review of Immunology, 1984
- G proteins and dual control of adenylate cyclaseCell, 1984
- A chemoattractant receptor on macrophages exists in two affinity states regulated by guanine nucleotides.The Journal of cell biology, 1984
- Effects of pertussis toxin treatment on the metabolism of rat adipocytes.Journal of Biological Chemistry, 1983
- Guanine nucleotides modulate the binding affinity of the oligopeptide chemoattractant receptor on human polymorphonuclear leukocytes.Journal of Clinical Investigation, 1983
- Transmethylation reactions regulate affinity and functional activity of chemotactic factor receptors on macrophagesCell, 1982
- Phosphate incorporation and Na, K‐ATPase activity in human red blood cell ghostsJournal of Cellular and Comparative Physiology, 1965