Interaction of Bradykinin with Angiotensin, Prostacyclin, and Nitric Oxide in Myocardial Preservationa
- 1 June 1999
- journal article
- review article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 874 (1) , 286-294
- https://doi.org/10.1111/j.1749-6632.1999.tb09244.x
Abstract
Abstract: This review focuses on the importance of bradykinin in myocardial preservation during ischemic arrest. Bradykinin is released from the heart spontaneously in response to ischemic stress, which may be viewed as a survival signal of the heart against ischemia. Bradykinin appears to function as a signaling molecule by controlling the release of other intracellular modulators, such as prostacyclins and nitric oxide, which also exert beneficial effects on the ischemic myocardium.Keywords
This publication has 53 references indexed in Scilit:
- Effects of Bradykinin and Endothelin-1 on the Calcium Homeostasis of Mammalian CellsPublished by Elsevier ,1995
- Cardioprotective effect of the angiotensin II type 1 receptor antagonist TCV-116 on ischemia-reperfusion injuryAmerican Heart Journal, 1994
- Captopril inhibits endothelin-1 secretion from endothelial cells through bradykinin.Hypertension, 1993
- Regulation of bradykinin-induced phosphoinositide turnover in cultured cerebellar astrocytes: possible role of protein kinase CNeurochemistry International, 1992
- Induction of endothelin-1 gene by angiotensin and vasopressin in endothelial cells.Hypertension, 1992
- Biochemical And Molecular Pharmacology Of Kinin ReceptorsAnnual Review of Pharmacology and Toxicology, 1992
- Ramiprilat enhances endothelial autacoid formation by inhibiting breakdown of endothelium-derived bradykinin.Hypertension, 1991
- Involvement of the Renin–Angiotensin System in Ischemic Damage and Reperfusion Arrhythmias in the Isolated Perfused Rat HeartJournal of Cardiovascular Pharmacology, 1991
- Evidence that phosphoinositide response is mediated by α1-adrenoceptor stimulation, but not linked with excitation-contraction coupling in cardiac muscleBiochemical and Biophysical Research Communications, 1986
- Limitation of experimental infarct size by an angiotensin-converting enzyme inhibitor.Circulation, 1982