The 5′HS4 core element of the human β-globin locus control region is required for high-level globin gene expression in definitive but not in primitive erythropoiesis 1 1Edited by M. Yaniv
- 1 September 2001
- journal article
- Published by Elsevier in Journal of Molecular Biology
- Vol. 312 (1) , 17-26
- https://doi.org/10.1006/jmbi.2001.4939
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- A deletion of the human beta-globin locus activation region causes a major alteration in chromatin structure and replication across the entire beta-globin locus.Genes & Development, 1990
- Gamma delta beta-thalassemia due to a de novo mutation deleting the 5' beta-globin gene activation-region hypersensitive sites.Proceedings of the National Academy of Sciences, 1989
- Molecular analysis of the human beta-globin locus activation region.Proceedings of the National Academy of Sciences, 1989
- A single erythroid-specific DNase I super-hypersensitive site activates high levels of human beta-globin gene expression in transgenic mice.Genes & Development, 1989
- The β-globin dominant control region activates homologous and heterologous promoters in a tissue-specific mannerCell, 1989
- Position-independent, high-level expression of the human β-globin gene in transgenic miceCell, 1987
- A developmentally stable chromatin structure in the human beta-globin gene cluster.Proceedings of the National Academy of Sciences, 1986
- The "beta-like-globin" gene domain in human erythroid cells.Proceedings of the National Academy of Sciences, 1985
- A distant gene deletion affects beta-globin gene function in an atypical gamma delta beta-thalassemia.Journal of Clinical Investigation, 1985
- β-Globin gene inactivation by DNA translocation in γβ-thalassaemiNature, 1983