Chronic Diarrhea Due to Excessive Bile Acid Synthesis and Not Defective Ileal Transport: A New Syndrome of Defective Fibroblast Growth Factor 19 Release
Open Access
- 10 August 2009
- journal article
- editorial
- Published by Elsevier in Clinical Gastroenterology and Hepatology
- Vol. 7 (11) , 1151-1154
- https://doi.org/10.1016/j.cgh.2009.07.026
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Bile acids: regulation of synthesisJournal of Lipid Research, 2009
- Bile acids activate fibroblast growth factor 19 signaling in human hepatocytes to inhibit cholesterol 7α‐hydroxylase gene expression†Hepatology, 2008
- Liver-specific Activities of FGF19 Require Klotho betaJournal of Biological Chemistry, 2007
- Molecular Insights into the Klotho-Dependent, Endocrine Mode of Action of Fibroblast Growth Factor 19 Subfamily MembersMolecular and Cellular Biology, 2007
- Normal or increased bile acid uptake in isolated mucosa from patients with bile acid malabsorptionEuropean Journal of Gastroenterology & Hepatology, 2006
- Bile Acid Malabsorption in Patients with Chronic Diarrhoea: Clinical Value of SeHCAT TestScandinavian Journal of Gastroenterology, 2003
- Absence of Dysfunctional Ileal Sodium?Bile Acid Cotransporter Gene Mutations in Patients with Adult-Onset Idiopathic Bile Acid MalabsorptionScandinavian Journal of Gastroenterology, 2001
- Primary bile acid diarrhoea without an ileal carrier defect: quantification of active bile acid transport across the ileal brush border membrane.Gut, 1991
- Is bile acid malabsorption underdiagnosed? An evaluation of accuracy of diagnosis by measurement of SeHCAT retention.BMJ, 1985
- Bile acid malabsorption and bile acid diarrhea in intestinal resectionDigestive Diseases and Sciences, 1982