Abstract
In 1862 William John Little, M.D., an orthopedic practitioner in London, described 47 children with what he termed spastic rigidity.1 His clear account leaves no doubt that he was speaking of the spastic form of the syndrome that we now call cerebral palsy. "Cerebral palsy" is a descriptive term for a collection of nonprogressive neuromotor disorders of central origin that become manifest early in life and are not the result of a recognized cerebral malformation. The main clinical feature is a lack of motor control, presenting most commonly as spasticity or, less commonly, as involuntary movements or incoordination. Any voluntary-muscle . . .

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