High levels of fatty acids delay the recoveryof intracellular pH and cardiac efficiency inpost-ischemic hearts by inhibiting glucose oxidation
- 8 February 2002
- journal article
- Published by Elsevier in Journal of the American College of Cardiology
- Vol. 39 (4) , 718-725
- https://doi.org/10.1016/s0735-1097(01)01803-4
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Preconditioning rabbit cardiomyocytes: role of pH, vacuolar proton ATPase, and apoptosis.Journal of Clinical Investigation, 1996
- Regulation of fatty acid oxidation in the mammalian heart in health and diseaseBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1994
- New Na(+)-H+ exchange inhibitor HOE 694 improves postischemic function and high-energy phosphate resynthesis and reduces Ca2+ overload in isolated perfused rabbit heart.Circulation, 1994
- Role of Na+/H+ exchange in cardiac physiology and pathophysiology: mediation of myocardial reperfusion injury by the pH paradoxCardiovascular Research, 1993
- Myocardial fatty acid oxidation during ischemia and reperfusionMolecular and Cellular Biochemistry, 1992
- Protons in ischemia: Where do they come from; Where do they go to?Journal of Molecular and Cellular Cardiology, 1991
- The pharmacology of dichloroacetateMetabolism, 1989
- Energy metabolism in reperfused heart muscle: Metabolic correlates to return of functionJournal of the American College of Cardiology, 1985
- Use of gated perfusion to study early effects of anoxia on cardiac energy metabolism: a new phosphorus-31 NMR methodBiochemistry, 1984
- Relationship Between Carbohydrate and Lipid Metabolism and the Energy Balance of Heart MuscleAnnual Review of Physiology, 1974