Vaccination with Glutamic Acid Decarboxylase Plasmid DNA Protects Mice from Spontaneous Autoimmune Diabetes and B7/CD28 Costimulation Circumvents That Protection
- 31 May 2001
- journal article
- Published by Elsevier in Clinical Immunology
- Vol. 99 (2) , 241-252
- https://doi.org/10.1006/clim.2001.5012
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- DNA immunization to prevent autoimmune diabetesJournal of Clinical Investigation, 1999
- Control of Autoimmune Diabetes in NOD Mice by GAD Expression or Suppression in β CellsScience, 1999
- TGF-β1 Alters APC Preference, Polarizing Islet Antigen Responses toward a Th2 PhenotypeImmunity, 1998
- GAD-reactive CD4+ Th1 cells induce diabetes in NOD/SCID mice.Journal of Clinical Investigation, 1998
- The Preferential Induction of a Th1 Immune Response by DNA-Based Immunization Is Mediated by the Immunostimulatory Effect of Plasmid DNACellular Immunology, 1997
- Immunomodulatory effects of a plasmid expressing B7‐2 on human immunodeficiency virus‐1‐specific cell‐mediated immunity induced by a plasmid encoding the viral antigenEuropean Journal of Immunology, 1997
- DNA vector constructs that prime hepatitis B surface antigen-specific cytotoxic T lymphocyte and antibody responses in mice after intramuscular injectionJournal of Immunological Methods, 1996
- The Roles of Costimulation and Fas in T Cell Apoptosis and Peripheral ToleranceImmunity, 1996
- Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic miceNature, 1993
- Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetesNature, 1993