Regulation of AP-1 gene transcription factor binding activity in the rat brain during nicotine dependence
- 12 April 1999
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 264 (1-3) , 21-24
- https://doi.org/10.1016/s0304-3940(99)00172-x
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Fos B mutant mice: Loss of chronic cocaine induction of Fos-related proteins and heightened sensitivity to cocaine’s psychomotor and rewarding effectsProceedings of the National Academy of Sciences, 1997
- Molecular and Cellular Aspects of Nicotine AbusePublished by Elsevier ,1996
- Acute repeated nicotine injections increase enkephalin and decrease AP-1 DNA binding activity in rat adrenal medullaMolecular Brain Research, 1995
- Neuronal nicotinic receptors: Molecular organization and regulationsNeuropharmacology, 1995
- Chronic electroconvulsive seizure (ECS) treatment results in expression of a long-lasting AP-1 complex in brain with altered composition and characteristicsJournal of Neuroscience, 1994
- Nicotine stimulates the expression of cFos protein in the parvocellular paraventricular nucleus and brainstem catecholaminergic regionsEndocrinology, 1993
- Regulation of immediate early gene expression and AP-1 binding in the rat nucleus accumbens by chronic cocaine.Proceedings of the National Academy of Sciences, 1992
- Nicotine binding and nicotinic receptor subunit RNA after chronic nicotine treatmentJournal of Neuroscience, 1992
- Calcium influx through nicotinic receptor in rat central neurons: Its relevance to cellular regulationNeuron, 1992
- Central actions of nicotineMedicinal Research Reviews, 1982