Chronic Ethanol Treatment Induces H2O2 Production Selectively in Pericentral Regions of the Liver Lobule
- 1 October 1992
- journal article
- Published by Wiley in Alcohol, Clinical and Experimental Research
- Vol. 16 (5) , 839-842
- https://doi.org/10.1111/j.1530-0277.1992.tb01878.x
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Regulation of urea synthesis in sublobular regions of the liver lobule by oxygenBiochimica et Biophysica Acta (BBA) - General Subjects, 1990
- Catalase‐dependent ethanol oxidation in perfused rat liverEuropean Journal of Biochemistry, 1988
- A new method for the isolation of fresh hepatocytes from periportal and pericentral regions of the liver lobuleBiochemical and Biophysical Research Communications, 1988
- Alcohol and the Liver: 1984 UpdateHepatology, 1984
- Is hypoxia involved in the mechanism of alcohol-induced liver injury?*1Fundamental and Applied Toxicology, 1984
- Periportal and pericentral pyridine nucleotide fluorescence from the surface of the perfused liver: evaluation of the hypothesis that chronic treatment with ethanol produces pericentral hypoxia.Proceedings of the National Academy of Sciences, 1982
- Increased Hepatic Oxygenation following Ethanol Administration in the BaboonExperimental Biology and Medicine, 1977
- Experimental alcohol-induced hepatic necrosis: suppression by propylthiouracil.Proceedings of the National Academy of Sciences, 1975
- High Blood Acetaldehyde Levels after Ethanol AdministrationNew England Journal of Medicine, 1975
- Interaction of Mixed‐Function Oxidation with Biosynthetic ProcessesEuropean Journal of Biochemistry, 1973