N-Methyl-d-aspartate receptor activation results in regulation of extracellular signal-regulated kinases by protein kinases and phosphatases in glutamate-induced neuronal apototic-like death
- 19 December 2000
- journal article
- Published by Elsevier in Brain Research
- Vol. 887 (2) , 285-292
- https://doi.org/10.1016/s0006-8993(00)03003-1
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Extracellular-signal-regulated kinase signalling in neuronsCurrent Opinion in Neurobiology, 1999
- Inactivation of Mitogen-Activated Protein Kinases by a Mammalian Tyrosine-Specific Phosphatase, PTPBR7Biochemical and Biophysical Research Communications, 1999
- N‐Methyl‐d‐Aspartate Receptor Desensitisation Is Neuroprotective by Inhibiting Glutamate‐Induced Apoptotic‐Like DeathJournal of Neurochemistry, 1998
- Calcium and neuronal deathPublished by Springer Nature ,1998
- Serum‐free B27/neurobasal medium supports differentiated growth of neurons from the striatum, substantia nigra, septum, cerebral cortex, cerebellum, and dentate gyrusJournal of Neuroscience Research, 1995
- Protein tyrosine kinase PYK2 involved in Ca2+-induced regulation of ion channel and MAP kinase functionsNature, 1995
- Transcriptional Regulation by Extracellular signals: Mechanisms and SpecificityCell, 1995
- Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activationCell, 1995
- Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications.Proceedings of the National Academy of Sciences, 1979
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970