Continuous glutamate leakage from brain cells is balanced by compensatory high-affinity reuptake transport
- 1 October 1987
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 81 (3) , 296-302
- https://doi.org/10.1016/0304-3940(87)90399-5
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Current views of the pathobiochemistry of epilepsyMolecular Aspects of Medicine, 1987
- Morphine inhibition of calcium fluxes, neurotransmitter release and protein and lipid phosphorylation in brain slices and synaptosomesBiochemical Pharmacology, 1986
- Ischemia-Induced Shift of Inhibitory and Excitatory Amino Acids from Intra- to Extracellular CompartmentsJournal of Cerebral Blood Flow & Metabolism, 1985
- Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral MicrodialysisJournal of Neurochemistry, 1984
- Differential Transmitter Release from Nerve Terminals Isolated from Basal Ganglia and Substantia NigraJournal of Neurochemistry, 1980
- Release of amino acids from the maturing cobalt-induced epileptic focusBrain Research, 1976
- Release of amino acids from the chronically-superfused mammalian cerebral cortexJournal of Neurochemistry, 1974
- DEPOLARIZING STIMULI AND THE RELEASE OF PHYSIOLOGICALLY ACTIVE AMINO ACIDS FROM SUSPENSIONS OF MAMMALIAN SYNAPTOSOMESJournal of Neurochemistry, 1973
- IONIC BASIS FOR THE DEPOLARIZATION OF CEREBRAL TISSUES BY EXCITATORY ACIDIC AMINO ACIDSJournal of Neurochemistry, 1966
- Continuous recording of changes in membrane potential in mammalian cerebral tissues in vitro; recovery after depolarization by added substancesThe Journal of Physiology, 1965