Acute Suppression of Inwardly Rectifying Kir2.1 Channels by Direct Tyrosine Kinase Phosphorylation
Open Access
- 1 December 1998
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (51) , 34063-34068
- https://doi.org/10.1074/jbc.273.51.34063
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Ontogeny of Gene Expression of Kir Channel Subunits in the RatMolecular and Cellular Neuroscience, 1997
- Association of Src Tyrosine Kinase with a Human Potassium Channel Mediated by SH3 DomainScience, 1996
- Role of c-Src Tyrosine Kinase in G Protein-coupled Receptorand Gβγ Subunit-mediated Activation of Mitogen-activated Protein KinasesJournal of Biological Chemistry, 1996
- The β-Adrenergic Receptor Is a Substrate for the Insulin Receptor Tyrosine KinaseJournal of Biological Chemistry, 1996
- The Trk family of neurotrophin receptorsJournal of Neurobiology, 1994
- Channel Regulation: Ion channel control by tyrosine phosphorylationCurrent Biology, 1994
- Tyrosine kinase-dependent suppression of a potassium channel by the G protein-coupled m1 muscarinic acetylcholine receptorCell, 1993
- Mode-switching of a voltage-gated cation channel is mediated by a protein kinase A-regulated tyrosine phosphataseNature, 1993
- Growth factor signaling by receptor tyrosine kinasesNeuron, 1992
- Human epidermal growth factor receptor cDNA sequence and aberrant expression of the amplified gene in A431 epidermoid carcinoma cellsNature, 1984