Radiation Induction of p53 in Cells from Nijmegen Breakage Syndrome Is Defective but Not Similar to Ataxia-Telangiectasia
- 1 January 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 242 (3) , 602-607
- https://doi.org/10.1006/bbrc.1997.7924
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 19 references indexed in Scilit:
- Genetic Mapping Using Microcell-Mediated Chromosome Transfer Suggests a Locus for Nijmegen Breakage Syndrome at Chromosome 8q21-24American Journal of Human Genetics, 1997
- Chromosomal sensitivity to clastogenic agents and cell cycle perturbations in Nijmegen Breakage Syndrome lymphoblastoid cell linesInternational Journal of Radiation Biology, 1997
- The Role of p53 in Cell Cycle RegulationPathology - Research and Practice, 1996
- Radiation-induced cell cycle arrest compromised by p21 deficiencyNature, 1995
- Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint controlCell, 1995
- Cell Cycle Control and CancerScience, 1994
- Nijmegen Breakage Syndrome: A Progress ReportInternational Journal of Radiation Biology, 1994
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasiaCell, 1992
- A NEW CHROMOSOMAL INSTABILITY DISORDER: THE NIJMEGEN BREAKAGE SYNDROMEActa Paediatrica, 1981