Inactivation ofp16/CDKN2 andp15/MTS2 genes in different histological types and clinical stages of primary ovarian tumors
Open Access
- 6 December 1996
- journal article
- research article
- Published by Wiley in International Journal of Cancer
- Vol. 69 (6) , 466-470
- https://doi.org/10.1002/(sici)1097-0215(19961220)69:6<466::aid-ijc8>3.0.co;2-2
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Genetic alterations distinguish different types of ovarian tumorsInternational Journal of Cancer, 1995
- 5′ CpG island methylation is associated with transcriptional silencing of the tumour suppressor p16/CDKN2/MTS1 in human cancersNature Medicine, 1995
- Alteration of the p53 Tumor Suppressor Gene Occurs Independently of K‐ras Activation and More Frequently in Serous Adenocarcinomas than in Other Common Epithelial Tumors of the Human OvaryJapanese Journal of Cancer Research, 1994
- Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinomaNature Genetics, 1994
- Germline p16 mutations in familial melanomaNature Genetics, 1994
- pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrestNature, 1994
- A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor TypesScience, 1994
- Early de novo ovarian carcinoma. A study of fourteen casesCancer, 1994
- Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancersNature, 1994
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993