Suppression of Kinesin Expression Disrupts Adenomatous Polyposis Coli (APC) Localization and Affects β-Catenin Turnover in Young Adult Mouse Colon (YAMC) Epithelial Cells
- 1 October 2002
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 280 (1) , 12-23
- https://doi.org/10.1006/excr.2002.5506
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Where is APC going?The Journal of cell biology, 2001
- Complex Formation of Adenomatous Polyposis Coli Gene Product and Axin Facilitates Glycogen Synthase Kinase-3β-dependent Phosphorylation of β-Catenin and Down-regulates β-CateninJournal of Biological Chemistry, 2000
- Identification and characterization of a 500-kb homozygously deleted region at 1p36.2-p36.3 in a neuroblastoma cell lineOncogene, 2000
- Downregulation of β-catenin by human Axin and its association with the APC tumor suppressor, β-catenin and GSK3βPublished by Elsevier ,1998
- Novel protein isoforms of the APC tumor suppressor in neural tissueOncogene, 1998
- Constitutive Transcriptional Activation by a β-Catenin-Tcf Complex in APC −/− Colon CarcinomaScience, 1997
- Binding of GSK3β to the APC-β-Catenin Complex and Regulation of Complex AssemblyScience, 1996
- Differential Display of Messenger Ribonucleic AcidNeurosurgery, 1995
- Altered homologous and heterologous gap‐junctional intercellular communication in primary human liver tumors associated with aberrant protein localization but not gene mutation of connexin 32International Journal of Cancer, 1994
- Suppression of kinesin expression in cultured hippocampal neurons using antisense oligonucleotides.The Journal of cell biology, 1992