Assessing disease onset and progression in the SOD1 mouse model of ALS
- 1 May 2003
- journal article
- Published by Wolters Kluwer Health in NeuroReport
- Vol. 14 (7) , 1051-1054
- https://doi.org/10.1097/00001756-200305230-00029
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Pathways to motor neuron degeneration in transgenic mouse modelsBiochimie, 2002
- Cyclooxygenase 2 inhibition protects motor neurons and prolongs survival in a transgenic mouse model of ALSAnnals of Neurology, 2002
- Transgenic mouse model for familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutationNeuropathology, 2001
- Neuroprotective effects of creatine in a transgenic animal model of amyotrophic lateral sclerosisNature Medicine, 1999
- Massive Mitochondrial Degeneration in Motor Neurons Triggers the Onset of Amyotrophic Lateral Sclerosis in Mice Expressing a Mutant SOD1Journal of Neuroscience, 1998
- Quantitative motor assessment in FALS miceNeuroReport, 1997
- A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase ( SOD1 ) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosisActa Neuropathologica, 1997
- Mice lacking both subunits of lysosomal β–hexosaminidase display gangliosidosis and mucopolysaccharidosisNature Genetics, 1996
- Benefit of vitamin E, riluzole, and gababapentin in a transgenic model of familial amyotrophic lateral sclerosisAnnals of Neurology, 1996
- Motor Neuron Degeneration in Mice that Express a Human Cu,Zn Superoxide Dismutase MutationScience, 1994