Our shifting understanding of the role of nitric oxide in autoimmune encephalomyelitis: a review

This publication has 66 references indexed in Scilit:

• Impairment of TNF-Receptor-1 Signaling but not Fas Signaling Diminishes T-Cell Apoptosis in Myelin Oligodendrocyte Glycoprotein Peptide-Induced Chronic Demyelinating Autoimmune Encephalomyelitis in Mice
  The American Journal of Pathology, 1999
• Nitric oxide is a potential down-regulating molecule in autoimmune disease: inhibition of nitric oxide production renders PVG rats highly susceptible to EAE
  Journal of Neuroimmunology, 1998
• The Multiplex Function of Nitric Oxide in (Auto)immunity
  The Journal of Experimental Medicine, 1998
• Time-course changes of nerve growth factor, corticotropin-releasing hormone, and nitric oxide synthase isoforms and their possible role in the development of inflammatory response in experimental allergic encephalomyelitis
  Proceedings of the National Academy of Sciences, 1997
• Inducible nitric oxide synthase gene expression and enzyme activity correlate with disease activity in murine experimental autoimmune encephalomyelitis
  Journal of Neuroimmunology, 1996
• Mechanisms of Immune Injury in Multiple Sclerosis
  Brain Pathology, 1996
• Aminoguanidine, an inhibitor of inducible nitric oxide synthase, ameliorates experimental autoimmune encephalomyelitis in SJL mice.
  Journal of Clinical Investigation, 1994
• Nitric oxide, an endothelial cell relaxation factor, inhibits neutrophil superoxide anion production via a direct action on the NADPH oxidase.
  Journal of Clinical Investigation, 1992
• Suppression of lymphocyte proliferation through the nitric oxide synthesizing pathway
  Journal of Surgical Research, 1991
• Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide.
  Proceedings of the National Academy of Sciences, 1990