Nonneural Nuclear Inclusions of Androgen Receptor Protein in Spinal and Bulbar Muscular Atrophy

This publication has 42 references indexed in Scilit:

• Intranuclear Neuronal Inclusions: A Common Pathogenic Mechanism for Glutamine-Repeat Neurodegenerative Diseases?
  Published by Elsevier ,1997
• Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures
  Nature, 1997
• The cerebellar leucine-rich acidic nuclear protein interacts with ataxin-1
  Nature, 1997
• Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in Brain
  Science, 1997
• Cloning of the SCA7 gene reveals a highly unstable CAG repeat expansion
  Nature Genetics, 1997
• Intranuclear Inclusions of Expanded Polyglutamine Protein in Spinocerebellar Ataxia Type 3
  Neuron, 1997
• A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes
  Cell, 1993
• Strong correlation between the number of CAG repeats in androgen receptor genes and the clinical onset of features of spinal and bulbar muscular atrophy
  Neurology, 1992
• Severity of X‐linked recessive bulbospinal neuronopathy correlates with size of the tandem cag repeat in androgen receptor gene
  Annals of Neurology, 1992
• Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy
  Nature, 1991