Inhibition of glycine oxidation by pyruvate, α-ketoglutarate, and branched-chain α-keto acids in rat liver mitochondria: Presence of interaction between the glycine cleavage system and α-keto acid dehydrogenase complexes
- 1 September 1986
- journal article
- research article
- Published by Elsevier in Archives of Biochemistry and Biophysics
- Vol. 249 (2) , 263-272
- https://doi.org/10.1016/0003-9861(86)90002-0
Abstract
No abstract availableThis publication has 46 references indexed in Scilit:
- Defective glycine cleavage system in nonketotic hyperglycinemia. Occurrence of a less active glycine decarboxylase and an abnormal aminomethyl carrier protein.Journal of Clinical Investigation, 1981
- Reduction of the level of the glycine cleavage system in the rat liver resulting from administration of dipropylacetic acid: An experimental approach to hyperglycinemiaArchives of Biochemistry and Biophysics, 1979
- Inhibition of glycine synthase by branched-chain α-keto acidsArchives of Biochemistry and Biophysics, 1978
- Mechanism of reversible glycine cleavage reaction in Arthrobacter globiformisArchives of Biochemistry and Biophysics, 1976
- Glycine metabolism by rat liver mitochondriaArchives of Biochemistry and Biophysics, 1974
- Glycine metabolism by rat liver mitochondriaArchives of Biochemistry and Biophysics, 1974
- Multiplicity and Origin of Isoenzymes of Lipoyl DehydrogenaseEuropean Journal of Biochemistry, 1972
- Glycine metabolism in rat liver mitochondriaArchives of Biochemistry and Biophysics, 1971
- DEFECTIVE PROPIONATE CARBOXYLATION IN KETOTIC HYPERGLYCINÆMIAThe Lancet, 1969
- The Structure of “Active Formaldehyde” (N5, N10 -Methylene Tetrahydrofolic Acid)1Journal of the American Chemical Society, 1960