Hot Spot Focusing of Somatic Hypermutation in MSH2-Deficient Mice Suggests Two Stages of Mutational Targeting

This publication has 22 references indexed in Scilit:

• Hypermutation of Immunoglobulin Genes in Memory B Cells of DNA Repair–deficient Mice
  The Journal of Experimental Medicine, 1998
• Analysis of the targeting of the hypermutational machinery and the impact of subsequent selection on the distribution of nucleotide changes in human V rearrangements
  Immunological Reviews, 1998
• Multiple sequences from downstream of the Jκ cluster can combine to recruit somatic hypermutation to a heterologous, upstream mutation domain
  European Journal of Immunology, 1998
• Hyperresponsive B Cells in CD22-Deficient Mice
  Science, 1996
• Clonal selection and learning in the antibody system
  Nature, 1996
• Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer
  Cell, 1995
• Hypermutation generating the sheep immunoglobulin repertoire is an antigen-independent process
  Cell, 1995
• Age-related decrease in the proportion of germinal center B cells from mouse Peyer's patches is accompanied by an accumulation of somatic mutations in their immunoglobulin genes
  European Journal of Immunology, 1994
• Mutation Drift and Repertoire Shift in the Maturation of the Immune Response
  Immunological Reviews, 1987
• Memory B cells at successive stages of differentiation. Affinity maturation and the role of IgD receptors
  The Journal of Experimental Medicine, 1980