Human Keratinocytes Regulate Their Expression of B7/BB-1 Antigen by a Unique, Calcium-Dependent Mechanism
- 1 May 1995
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 104 (5) , 763-767
- https://doi.org/10.1111/1523-1747.ep12606982
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- In Situ Expression of a B7-Like Adhesion Molecule on Keratinocytes from Human EpidermisJournal of Investigative Dermatology, 1993
- Accessory and Alloantigen-Presenting Cell Functions of A431 Keratinocytes That Stably Express the B7 AntigenCellular Immunology, 1993
- Non-antigen signals for B-cell growth and differentiation to antibody secretionCurrent Opinion in Immunology, 1993
- Phorbol-12-Myristate-13-Acetate-Treated Human Keratinocytes Express B7-Like Molecules That Serve a Costimulatory Role in T-Cell ActivationJournal of Investigative Dermatology, 1993
- The role of cell division in the induction of clonal anergyImmunology Today, 1992
- Selective induction of B7/BB-1 on interferon-γ stimulated monocytes: A potential mechanism for amplification of T cell activation through the CD28 pathwayCellular Immunology, 1991
- CTLA-4 is a second receptor for the B cell activation antigen B7.The Journal of Experimental Medicine, 1991
- Role of the CD28 receptor in T-cell activationImmunology Today, 1990
- Clonal Expansion Versus Functional Clonal Inactivation: A Costimulatory Signalling Pathway Determines the Outcome of T Cell Antigen Receptor OccupancyAnnual Review of Immunology, 1989
- Calcium regulation of growth and differentiation of mouse epidermal cells in culturePublished by Elsevier ,1980