Failure of the peroxisome proliferator WY-14,643 to initiate growth-selectible foci in rat liver
- 1 May 1989
- journal article
- research article
- Published by Elsevier in Toxicology
- Vol. 56 (1) , 1-7
- https://doi.org/10.1016/0300-483x(89)90207-2
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- 32P-Postlabeling analysis of peroxisome proliferator-DNA adduct formation in rat liver in vivo and hepatocytes in vitroCarcinogenesis: Integrative Cancer Research, 1985
- Lack of covalent binding of peroxisome proliferators nafenopin and WY-14 643 to DNA in vivo and in vitroToxicology Letters, 1984
- Effect of hypolipidemic peroxisome proliferators on unscheduled DNA synthesis in cultured hepatocytes and on mutagenesis in SalmonellaCancer Letters, 1984
- Investigation of the potential for binding of di(2-ethylhexyl) phthalate (DEHP) and di(2-ethylhexyl) adipate (DEHA) to liver DNA in vivoToxicology and Applied Pharmacology, 1984
- Lack of genotoxic activity of di(2-ethylhexyl)phthalate (DEHP) in rat and human hepatocytesCarcinogenesis: Integrative Cancer Research, 1984
- Carcinogenesis by Hepatic Peroxisome Proliferators: Evaluation of the Risk of Hypolipidemic Drugs and Industrial Plasticizers to HumansCRC Critical Reviews in Toxicology, 1983
- Lack of covalent binding to rat liver DNA of the hypolipidemic drugs clofibrate and fenofibrateToxicology Letters, 1981
- Hypolipidaemic hepatic peroxisome proliferators form a novel class of chemical carcinogensNature, 1980
- Tumorigenicity of the hypolipidaemic peroxisome proliferator ethyl-α-p-chlorophenoxyisobutyrate (clofibrate) in ratsBritish Journal of Cancer, 1979
- New principle for the analysis of chemical carcinogenesisNature, 1976