Ataxia-telangiectasia-mutated (ATM) and NBS1-dependent Phosphorylation of Chk1 on Ser-317 in Response to Ionizing Radiation
Open Access
- 1 April 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (17) , 14806-14811
- https://doi.org/10.1074/jbc.m210862200
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Determination of Substrate Motifs for Human Chk1 and hCds1/Chk2 by the Oriented Peptide Library ApproachJournal of Biological Chemistry, 2002
- The DNA damage-dependent intra–S phase checkpoint is regulated by parallel pathwaysNature Genetics, 2002
- Dominant Negative ATM Mutations in Breast Cancer FamiliesJNCI Journal of the National Cancer Institute, 2002
- ATR and ATRIP: Partners in Checkpoint SignalingScience, 2001
- DNA Damage-dependent and -independent Phosphorylation of the hRad9 Checkpoint ProteinJournal of Biological Chemistry, 2001
- ATR-Mediated Checkpoint Pathways Regulate Phosphorylation and Activation of Human Chk1Molecular and Cellular Biology, 2001
- Involvement of Brca1 in S-Phase and G2-Phase Checkpoints after Ionizing IrradiationMolecular and Cellular Biology, 2001
- P53 Binding Protein 1 (53bp1) Is an Early Participant in the Cellular Response to DNA Double-Strand BreaksThe Journal of cell biology, 2000
- Recombinant ATM protein complements the cellular A-T phenotypeOncogene, 1997
- Atm-Deficient Mice: A Paradigm of Ataxia TelangiectasiaCell, 1996