Insulin secretory defects and impaired islet architecture in pancreatic β-cell-specific STAT3 knockout mice
- 1 July 2004
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 319 (4) , 1159-1170
- https://doi.org/10.1016/j.bbrc.2004.05.095
Abstract
No abstract availableKeywords
Funding Information
- Ministerio de Educación, Cultura y Deporte
- Juvenile Diabetes Research Foundation International
- Japan Society for the Promotion of Science
This publication has 49 references indexed in Scilit:
- STAT3 signalling is required for leptin regulation of energy balance but not reproductionNature, 2003
- Adenovirus-mediated Hepatocyte Growth Factor Expression in Mouse Islets Improves Pancreatic Islet Transplant Performance and Reduces Beta Cell DeathJournal of Biological Chemistry, 2003
- Pancreatic β-cell growth and survival – a role in obesity-linked type 2 diabetes?Trends in Molecular Medicine, 2002
- Dual Roles for Glucokinase in Glucose Homeostasis as Determined by Liver and Pancreatic β Cell-specific Gene Knock-outs Using Cre RecombinaseJournal of Biological Chemistry, 1999
- Early diabetes and abnormal postnatal pancreatic islet development in mice lacking Glut-2Nature Genetics, 1997
- Expression of Heparin-binding Epidermal Growth Factor-like Growth Factor during Pancreas DevelopmentJournal of Biological Chemistry, 1997
- Efficient Regulation of Gene Expression by Adenovirus Vector-Mediated Delivery of the CRE RecombinaseBiochemical and Biophysical Research Communications, 1995
- Pancreatic β-Cell-specific Targeted Disruption of Glucokinase GeneJournal of Biological Chemistry, 1995
- Transcriptional Activation of the IL-6 Response Element in the junB Promoter Is Mediated by Multiple Stat Family ProteinsBiochemical and Biophysical Research Communications, 1994
- Stat3: a STAT Family Member Activated by Tyrosine Phosphorylation in Response to Epidermal Growth Factor and Interleukin-6Science, 1994