Failure to activate NF-κB promotes apoptosis of retinal ganglion cells following optic nerve transection
- 10 November 2000
- journal article
- Published by Elsevier in Brain Research
- Vol. 883 (1) , 60-68
- https://doi.org/10.1016/s0006-8993(00)02886-9
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Nitric Oxide Synthase Inhibition Delays Axonal Degeneration and Promotes the Survival of Axotomized Retinal Ganglion CellsExperimental Neurology, 1999
- Multiple signals converging on NF-κBPublished by Elsevier ,1999
- Retinal Ganglion Cells Lose Trophic Responsiveness after AxotomyNeuron, 1999
- Possible role of NF-κB and p53 in the glutamate-induced pro-apoptotic neuronal pathwayCell Death & Differentiation, 1999
- NF-κB Antiapoptosis: Induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to Suppress Caspase-8 ActivationScience, 1998
- Neuronal Cell DeathNeuron, 1998
- Drug-Induced Neuroprotection From Global Ischemia Is Associated With Prevention of Persistent but Not Transient Activation of Nuclear Factor-κB in RatsStroke, 1998
- Sulfasalazine: a potent and specific inhibitor of nuclear factor kappa B.Journal of Clinical Investigation, 1998
- NF-κB Activation: The IκB Kinase Revealed?Cell, 1997
- Rat retinal müller cells express Thy‐1 following neuronal cell deathGlia, 1995