Aryl Hydrocarbon Receptor (AhR)-Mediated Induction of Xanthine Oxidase/Xanthine Dehydrogenase Activity by 2,3,7,8-Tetrachlorodibenzo-p-dioxin
- 1 March 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 281 (5) , 1093-1099
- https://doi.org/10.1006/bbrc.2001.4464
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- Loss of teratogenic response to 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin (TCDD) in mice lacking the Ah (dioxin) receptorGenes to Cells, 1997
- Xanthine oxidase activity and free radical generation in patients with sepsis syndromeCritical Care Medicine, 1996
- Aryl-hydrocarbon Receptor-Deficient Mice Are Resistant to 2,3,7,8-Tetrachlorodibenzo-p-dioxin-Induced ToxicityToxicology and Applied Pharmacology, 1996
- Oxidative stress induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin is mediated by the aryl hydrocarbon (Ah) receptor complexToxicology, 1994
- Mechanisms of Reperfusion InjuryThe Lancet Healthy Longevity, 1994
- Oxidative stress induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)Free Radical Biology & Medicine, 1990
- 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD)-induced lipid peroxidation in genetically responsive and non-responsive miceArchives of Environmental Contamination and Toxicology, 1988
- 3 Molybdenum Hydroxylases: Biological Distribution and Substrate-Inhibitor SpecificityPublished by Elsevier ,1987
- 2,3,7,8-Tetrachlorodibenzo-p-Dioxin and Related Halogenated Aromatic Hydrocarbons: Examination of the Mechanism of ToxicityAnnual Review of Pharmacology and Toxicology, 1982
- Toxicity of 2, 3, 7, 8-Tetrachlorodibenzo-p-dioxin (TCDD)Drug Metabolism Reviews, 1982