[43] Detection of polyglutamine aggregation in mouse models
- 1 January 1999
- book chapter
- Published by Elsevier
- Vol. 309, 687-701
- https://doi.org/10.1016/s0076-6879(99)09045-x
Abstract
No abstract availableThis publication has 15 references indexed in Scilit:
- Transgenic Mice in the Study of Polyglutamine Repeat Expansion DiseasesBrain Pathology, 1998
- Spinocerebellar ataxia type 7 (SCA7): a neurodegenerative disorder with neuronal intranuclear inclusionsHuman Molecular Genetics, 1998
- Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat LengthNeurobiology of Disease, 1998
- Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structuresNature, 1997
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997
- Intranuclear Inclusions of Expanded Polyglutamine Protein in Spinocerebellar Ataxia Type 3Neuron, 1997
- Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD MutationCell, 1997
- Huntingtin-Encoded Polyglutamine Expansions Form Amyloid-like Protein Aggregates In Vitro and In VivoCell, 1997
- Instability of highly expanded CAG repeats in mice transgenic for the Huntington's disease mutationNature Genetics, 1997
- Exon 1 of the HD Gene with an Expanded CAG Repeat Is Sufficient to Cause a Progressive Neurological Phenotype in Transgenic MiceCell, 1996