Ubiquitination capabilities in response to neocarzinostatin and H2O2 stress in cell lines from patients with ataxia-telangiectasia
- 25 June 2002
- journal article
- research article
- Published by Springer Nature in Oncogene
- Vol. 21 (28) , 4363-4373
- https://doi.org/10.1038/sj.onc.1205557
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 57 references indexed in Scilit:
- The Ataxia telangiectasia Gene Product Is Required for Oxidative Stress-induced G1 and G2Checkpoint Function in Human FibroblastsJournal of Biological Chemistry, 2001
- Dopaminergic Loss and Inclusion Body Formation in α-Synuclein Mice: Implications for Neurodegenerative DisordersScience, 2000
- ATM: A mediator of multiple responses to genotoxic stressOncogene, 1999
- Ataxia telangiectasia heterozygotes and patients display increased fluidity and decrease in contents of sulfhydryl groups in red blood cell membranesBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1996
- Protein ubiquitination involving an E1–E2–E3 enzyme ubiquitin thioester cascadeNature, 1995
- Cellular Sensitivity to Oxidative Stress in the Photosensitivity Dermatitis/Actinic Reticuloid SyndromeJournal of Investigative Dermatology, 1994
- Ubiquitin and the molecular pathology of chronic degenerative diseasesThe Journal of Pathology, 1991
- Neocarzinostatin: Selective tryptophan oxidation and neocarzinostatin-chromophore binding to apo-neocarzinostatin.CHEMICAL & PHARMACEUTICAL BULLETIN, 1991
- Red Light-Induced Accumulation of Ubiquitin-Phytochrome Conjugates in Both Monocots and DicotsPlant Physiology, 1989
- Reduced inhibition of replicon initiation and chain elongation by neocarzinostatin in skin fibroblasts from patients with ataxia telangiectasiaBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1982