Differential effects of apamin on Ca2+-dependent K+ currents in bullfrog sympathetic ganglion cells

This publication has 16 references indexed in Scilit:

• Pharmacological evidence for two types of Ca2+-dependent K+-conductance in bullfrog sympathetic ganglion cells
  Neuroscience Research, 1985
• A calcium‐activated chloride current generates the after‐depolarization of rat sensory neurones in culture.
  The Journal of Physiology, 1985
• (+)-Tubocurarine blocks the Ca2+-dependent K+-channel of the bullfrog sympathetic ganglion cell
  Brain Research, 1984
• The coexistence in rat muscle cells of two distinct classes of Ca2+-dependent K+ channels with different pharmacological properties and different physiological functions
  Biochemical and Biophysical Research Communications, 1984
• Action potential repolarization may involve a transient, Ca2+ -sensitive outward current in a vertebrate neurone
  Nature, 1982
• Intracellular Ca2+ activates a fast voltage-sensitive K+ current in vertebrate sympathetic neurones
  Nature, 1982
• Release of calcium ions linked to the activation of potassium conductance in a caffeine‐treated sympathetic neurone.
  The Journal of Physiology, 1980
• Apamin blocks certain neurotransmitter-induced increases in potassium permeability
  Nature, 1979
• Spike after-hyperpolarisation of a sympathetic neurone is calcium sensitive and is potentiated by theophylline
  Nature, 1976
• CALCIUM IONS AND THE POST-TETANIC HYPERPOLARIZATION OF BULLFROG SYMPATHETIC GANGLION CELLS
  The Japanese Journal of Physiology, 1974