Two Posttranscriptional Pathways That Regulate p21Cip1/Waf1/Sdi1Are Identified by HPV16-E6 Interaction and Correlate with Life Span and Cellular Senescence
- 1 February 1999
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 247 (1) , 168-175
- https://doi.org/10.1006/excr.1998.4345
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Human Papillomavirus E6 and E7: Proteins which deregulate the cell cycleBioEssays, 1995
- p53-dependent G1 arrest involves pRB-related proteins and is disrupted by the human papillomavirus 16 E7 oncoprotein.Proceedings of the National Academy of Sciences, 1994
- Growth arrest by induction of p53 in DNA damaged keratinocytes is bypassed by human papillomavirus 16 E7.Proceedings of the National Academy of Sciences, 1994
- Human papillomavirus 16 E6 expression disrupts the p53-mediated cellular response to DNA damage.Proceedings of the National Academy of Sciences, 1993
- The two-stage mechanism controlling cellular senescence and immortalizationExperimental Gerontology, 1992
- Viruses in Human CancersScience, 1991
- A role for both RB and p53 in the regulation of human cellular senescence*1Experimental Cell Research, 1991
- Reversible cellular senescence: implications for immortalization of normal human diploid fibroblasts.Molecular and Cellular Biology, 1989
- Human Papillomaviruses and CarcinomasPublished by Elsevier ,1989
- Progress of aging in human diploid cells transformed with a tsA mutant of simian virus 40Experimental Cell Research, 1984