Transformation by ras oncogene induces nuclear shift of protein kinase C

This publication has 29 references indexed in Scilit:

• Rapid activation by erythropoietin of protein kinase C in nuclei of erythroid progenitor cells
  Biochemical and Biophysical Research Communications, 1990
• Potent selective inhibitors of protein kinase C
  FEBS Letters, 1989
• A mutant protein kinase C that can transform fibroblasts
  Nature, 1989
• Synthesis of diacylglycerol de novo is responsible for permanent activation and down-regulation of protein kinase C in transformed cells
  Biochemical and Biophysical Research Communications, 1989
• Signal transduction in EJ‐H‐ras‐transformed cells: De novo synthesis of diacylglycerol and subversion of agonist‐stimulated inositol lipid metabolism
  FEBS Letters, 1989
• Ia binding ligands and cAMP stimulate nuclear translocation of PKC in B lymphocytes
  Nature, 1987
• Point-mutated P21 couples a muscarinic receptor to calcium channels and polyphosphoinositide hydrolysis
  Biochemical and Biophysical Research Communications, 1986
• Normal p21N-ras couples bombesin and other growth factor receptors to inositol phosphate production
  Nature, 1986
• Studies and Perspectives of Protein Kinase C
  Science, 1986
• Transformation of balb3T3 cells with EJ/T24/H-Ras oncogene inhibits adenylate cyclase response to β-adrenergic agonist while increases muscarinic receptor dependent hydrolysis of inositol lipids
  Biochemical and Biophysical Research Communications, 1985